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Interestingly, selenium deficiency in rodents was found to have little impact on DIO activities as it appears that selenium is being supplied in priority for adequate synthesis of DIOs at the expense of other selenoenzymes (10)

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These modifications, including phosphorylation, acetylation, ubiquitination, SUMOylation, glycosylation, and PARylation, not only regulate NRF2s activity but also affect its interactions with other proteins, stability, and its role in critical cellular signaling pathways

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